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Future research should explore not only who responds to GLP-1 RA pharmacotherapy, but also how to maintain progress once medication ends, especially in vulnerable subgroups

High doses of TNF- lead to reduced muscle cell differentiation in human and mouse muscle cells [6,7] and cause myotube (in vitro) and myofibre (in vivo) atrophy in animals [8,9], suggesting a predominant role for pro-inflammatory cytokine in reduced muscle regeneration, and thus a contributor to atrophy

[Crossref] [Google Scholar] [PubMed] Iwaki T, Urano T, Umemura K

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Large-scale outcome trials involve substantial investment, lengthy follow-up periods, and extensive safety evaluations before regulatory approval