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Combination of prolonged water fasting and GLP-1 for refractory morbid obesity: Case report

Linus Pauling Institute Oregon State University Updated in August 2015 by: Barbara Delage, Ph.D

A combination of prolonged oxidative damage and hepatocellular proliferation is postulated to be responsible for liver tumor development, with tumor development primarily a result of enhanced growth of preneoplastic foci

Importantly, these pathological alterationsindicative of mitochondrial swelling and metabolic dysfunctionwere significantly attenuated by LEO treatment at both 30 and 60 mg/kg doses, with high-dose administration restoring near-normal ultrastructure

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GLP-1 medications mimic the natural action of the gut hormone glucagon-like peptide-1 (GLP-1), which regulates blood sugar levels and appetite