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Thus, while endocardial endothelial cells represent a plausible target for the antiinflammatory actions of GLP-1 medicines locally in both species, these medicines may also exert cardioprotective effects directly on human cardiomyocytes

The mechanism does not involve the kidneys dumping glucose into urine

In: FulopTFranceschiCHirokawaKPawelecGeditors

Association of GLP-1 Receptor Agonist Use With Risk of Gallbladder and Biliary Diseases [full text]

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