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IL-4 promotes asymmetric dimethylarginine accumulation, oxo-nitrative stress, and hypoxic response-induced mitochondrial loss in airway epithelial cells

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Early studies pointed to the plasma membrane as the primary execution site, supported by findings that FSP1, as described above, exerts its antiferroptotic function through the reduction of plasma membrane ubiquinone (CoQ 10 ) 16,17