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This oxidative damage severely impairs the mitochondria's ability to produce adenosine triphosphate (ATP), the currency of cellular energy

Malondialdehyde (MDA) and Hydrogen peroxide (H 2 O 2 ) concentrations The accumulation of MDA because of lipid peroxidation was assessed by the Thiobarbituric acid (TBA) according to Wang et al

OMM proteins such as NIX, FUNDC1, and BNIP3 can act as mitophagy-related receptors, relying on their LIR domain to bind with LC3, thereby directing dysfunctional mitochondria to autophagosomes for degradation via receptor-mediated mitophagy [71]