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Exenatide activated eNOS and NO production in endothelial cells, induced dose-dependent vasorelaxation, and mitigated high-glucose or lipid-induced endothelial dysfunction in arterioles ex vivo, effects which were reduced by AMPK (AMP-activated protein kinase) inhibition [86]

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doi: 10.1136/gut.38.6.916 23 RanganathLNorrisFMorganLWrightJMarksV

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